If you reside in an apartment or a house, or you just want to keep your home free of EMFs There are a variety of methods to reduce exposure. One of the most effective is to limit your use of electronic devices. It is also possible to use EMF blocker paint to block EMF radiation from reaching your home. Another easy way to protect your house against EMF radiation would be to install a RF shielding canopy. It is a type made of net which contains EMF shielding. It's used to block EMFs from entering rooms. Another option is to have your home fitted with an enclosure that is conductive. These devices are called Faraday cages.
Several studies have shown how the non-ionizing energy of RF can cause antiproliferative properties in HCC cells. The mechanism of AM RF EMF's anticancer activity in vitro is believed to involve down-regulation the cancer stem cell. This may account for the long-term responses seen in patients suffering from advanced HCC. However, the mechanism of AM EMF's impact on patients with cancer is not yet clear.
Aspects from AM RF EMFs on HCC tumor growth in vivo was studied in mice. emf blocking were split into three groups. The first group was not exposed to RF EMF. emf blockers of participants was subjected to RF EMF at frequencies similar to the one used by humans. In the third, they were subjected to RF EMF at HCC-specific modulation frequencies. The effect of HCCMF on tumours was assessed against the effect of RCF. The results showed that the tumors treated by HCCMF showed significant shrinkage. However, the tumors treated with RCF didn't show evidence of shrinkage of the tumor.
The mechanism behind tumor-specific AM RF EMF could be based on the fact that tumor cells require Cav3*2 T-type voltage calcium channels for proliferation and down-regulation. AM RF EMF's antiproliferative effect in HCC cells is mediated by CACNA1H the protein which regulates the Ca2+ influx specific to tumors. The results suggest that CACNA1H could have more broader implications for treatment and diagnosis of different cancers.
The tumors in the controls were never exposed to RF EMF, and were fed a normal diet of mice. The tumors in HCCMF HCCMF group were injected with Huh7 cells after they were 5 to 7 weeks old. The tumors were then killed when they showed excessive burden.
The tumours from the three groups showed distinct growth curves. The tumours treated with HCCMF showed a significant decrease in the size of the tumour after eight weeks. However, the tumors treated with RCF showed no signs of shrinkage. The difference was significant. The tumors treated by RCF were able to show necrosis, which is common when tumors are exposed to RCF. It is possible that the necrosis was caused by the lack of oxygen in larger tumors.
In emf blocker , the results indicate an AM-RF EMF is a powerful source of anticancer effects in vitro and in vivo. A number of studies have proven that AM RF EMF produces measurable reduction in tumours in HCC patients. There is a possibility that AM RF EMF produces these effects due to CACNA1H, a protein involved in tissue-specific Ca2+ influx. Additionally, AM RF EMF may have a long-lasting influence on the growth of HCC tumors in living tissue.