If you reside in an apartment or house or simply would like to ensure your home is free from EMFs There are a variety of ways you can reduce exposure. One of the most effective is to restrict your electronic device use. You can also turn to EMF block paint to prevent EMF radiation from reaching your home. Another easy way to protect your home from EMF radiations is to put up a RF shielding canopy. It is a type of net that contains EMF shielding and is used to prevent EMFs from entering rooms. Click here is to have your home equipped with an electrical enclosure. These devices are called Faraday cages.
Several studies have shown how the non-ionizing energy of RF produces antiproliferative effects in HCC cells. The mechanism behind AM RF EMF's anticancer activity in vitro is believed to be based on the downregulation of cancer stem cells. This could explain the long-term responses seen in some patients with advanced HCC. However, the mechanism of AM EMF's impact on patients with cancer is not clear.
The effects from AM electromagnetic fields (RFEM) on HCC tumor growth in vivo were studied in mice. The tumours were divided into 3 groups. First, the group that was unaffected RF EMF. Second group members were exposed to RF EMF at the same frequency to that used in humans. In the third, they were subjected RF EMF with HCC-specific modulation frequencies. The effect of HCCMF on tumours was assessed against the effect of RCF. The results revealed that tumors treated by HCCMF showed significant shrinkage. However, the tumors treated with RCF didn't show evidence of shrinkage in the tumour.
The mechanism behind cancer-specific AM RF EMF may be based on the fact that tumour cells require Cav3*2 voltage calcium channels for proliferation and down-regulation. AM RF EMF's antiproliferative effects in HCC cells is caused through CACNA1H, a protein that is responsible for the influx of Ca2+ specific to tumours. The results suggest that CACNA1H could have more broader implications for the treatment and diagnosis of different cancers.
The tumours in the control group were not exposed EMF from RF, and fed a normal mouse diet. The tumours in those in the HCCMF group were treated with Huh7 cells when they were five-seven weeks old. The tumors were then killed after they had a high burden.
The tumours from the three groups showed distinct growth curves. The tumors treated with HCCMF had a significant reduction in tumour size after 8 weeks. However, the tumours treated with RCF showed no shrinkage. The difference was highly significant. The tumors treated with RCF showed necrosis that is common in tumors that have been exposed to RCF. There is a possibility that the necrosis was caused by the lack of oxygen in larger tumours.
In sum, the results suggest the fact that AM EMF is a powerful source of anticancer effects in vitro and in live. A number of studies have proven it is true that AM RF EMF produces measurable reduction in tumours in HCC patients. It is possible that AM RF EMF triggers these effects through CACNA1H, a protein involved in the process of tissue-specific Ca2+ influx. Additionally, emf neutralizer may cause a lasting impact on the growth of HCC tumors in living tissue.